Cell Signaling Technology

PathScan® Phospho-mTOR (Ser2448) Sandwich ELISA Kit

#7976 · elisa · $66,129,740

100

Citing papers

5.7k

Combined citations

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Research areas

MicroRNA in disease regulation12 Immune Response and Inflammation8 Autophagy in Disease and Therapy8 NF-κB Signaling Pathways7 PI3K/AKT/mTOR signaling in cancer6 Circular RNAs in diseases5 Cancer Cells and Metastasis5 Cellular transport and secretion5

Papers citing this product

1

The two-pore channel TPCN2 mediates NAADP-dependent Ca2+-release from lysosomal stores

Pflügers Archiv - European Journal of Physiology·2009·271 cit.·DOI

2

Exercise training modulates the gut microbiota profile and impairs inflammatory signaling pathways in obese children

Experimental & Molecular Medicine·2020·210 cit.·DOI

3

A chalcone-related small molecule that induces methuosis, a novel form of non-apoptotic cell death, in glioblastoma cells

Molecular Cancer·2011·170 cit.·DOI

4

ERK3 signals through SRC-3 coactivator to promote human lung cancer cell invasion

Journal of Clinical Investigation·2012·168 cit.·DOI

5

Rab18 Binds to Hepatitis C Virus NS5A and Promotes Interaction between Sites of Viral Replication and Lipid Droplets

PLoS Pathogens·2013·154 cit.·DOI

6

Targeting Cullin-RING ligases by MLN4924 induces autophagy via modulating the HIF1-REDD1-TSC1-mTORC1-DEPTOR axis

Cell Death and Disease·2012·141 cit.·DOI

7

Isocitrate Dehydrogenase (IDH) Mutations Promote a Reversible ZEB1/MicroRNA (miR)-200-dependent Epithelial-Mesenchymal Transition (EMT)

Journal of Biological Chemistry·2012·140 cit.·DOI

8

MicroRNAs Link Estrogen Receptor Alpha Status and Dicer Levels in Breast Cancer

Hormones and Cancer·2010·134 cit.·DOI

9

Type IV Secretion-Dependent Activation of Host MAP Kinases Induces an Increased Proinflammatory Cytokine Response to Legionella pneumophila

PLoS Pathogens·2008·128 cit.·DOI

10

Clathrin- and Dynamin-Dependent Endocytic Pathway Regulates Muramyl Dipeptide Internalization and NOD2 Activation

The Journal of Immunology·2009·125 cit.·DOI

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