Cell Signaling Technology

Exportin 5 (D7W6W) Rabbit Monoclonal Antibody

#12565 · $161

100
Citing papers
5.4k
Combined citations
Supplier page Find this product

Research areas

Cancer-related molecular mechanisms research10Neuroinflammation and Neurodegeneration Mechanisms9MicroRNA in disease regulation9Growth Hormone and Insulin-like Growth Factors7Neurogenesis and neuroplasticity mechanisms7Genetics and Neurodevelopmental Disorders6Nerve injury and regeneration6Cellular transport and secretion6

Papers citing this product

1
Microglia are an essential component of the neuroprotective scar that forms after spinal cord injury
Nature Communications·2019·648 cit.·DOI
2
Supermeres are functional extracellular nanoparticles replete with disease biomarkers and therapeutic targets
Nature Cell Biology·2021·399 cit.·DOI
3
Rett Syndrome Microglia Damage Dendrites and Synapses by the Elevated Release of Glutamate
Journal of Neuroscience·2010·347 cit.·DOI
4
Reduced AKT/mTOR signaling and protein synthesis dysregulation in a Rett syndrome animal model
Human Molecular Genetics·2011·223 cit.·DOI
5
Neutrophils Are Critical for Myelin Removal in a Peripheral Nerve Injury Model of Wallerian Degeneration
Journal of Neuroscience·2017·190 cit.·DOI
6
Phosphorylation of ULK1 affects autophagosome fusion and links chaperone-mediated autophagy to macroautophagy
Nature Communications·2018·190 cit.·DOI
7
MeCP2-Mediated Transcription Repression in the Basolateral Amygdala May Underlie Heightened Anxiety in a Mouse Model of Rett Syndrome
Journal of Neuroscience·2009·138 cit.·DOI
8
Loss of MeCP2 From Forebrain Excitatory Neurons Leads to Cortical Hyperexcitation and Seizures
Journal of Neuroscience·2014·129 cit.·DOI
9
Vascular microRNA-204 is remotely governed by the microbiome and impairs endothelium-dependent vasorelaxation by downregulating Sirtuin1
Nature Communications·2016·119 cit.·DOI
10
Conditional deletion of Mecp2 in parvalbumin-expressing GABAergic cells results in the absence of critical period plasticity
Nature Communications·2014·114 cit.·DOI