Cell Signaling Technology

DHX29 (D19E10) Rabbit Monoclonal Antibody

#5926 · $161

100
Citing papers
7.8k
Combined citations
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Research areas

Immune Cell Function and Interaction8Autophagy in Disease and Therapy6T-cell and B-cell Immunology6Cancer Cells and Metastasis5Adipose Tissue and Metabolism5Endoplasmic Reticulum Stress and Disease5Estrogen and related hormone effects5Genomics, phytochemicals, and oxidative stress5

Papers citing this product

1
Resveratrol Inhibits Pancreatic Cancer Stem Cell Characteristics in Human and KrasG12D Transgenic Mice by Inhibiting Pluripotency Maintaining Factors and Epithelial-Mesenchymal Transition
PLoS ONE·2011·301 cit.·DOI
2
The Synthetic Triterpenoids, CDDO and CDDO-Imidazolide, Are Potent Inducers of Heme Oxygenase-1 and Nrf2/ARE Signaling
Cancer Research·2005·283 cit.·DOI
3
Effective delivery of large genes to the retina by dual AAV vectors
EMBO Molecular Medicine·2013·258 cit.·DOI
4
Triterpenoid CDDO-Me Blocks the NF-κB Pathway by Direct Inhibition of IKKβ on Cys-179
Journal of Biological Chemistry·2006·232 cit.·DOI
5
Mcl-1 Is a Key Regulator of Apoptosis during CNS Development and after DNA Damage
Journal of Neuroscience·2008·184 cit.·DOI
6
Zinc Transporter ZIP14 Functions in Hepatic Zinc, Iron and Glucose Homeostasis during the Innate Immune Response (Endotoxemia)
PLoS ONE·2012·183 cit.·DOI
7
Silver Nanoparticle-Induced Autophagic-Lysosomal Disruption and NLRP3-Inflammasome Activation in HepG2 Cells Is Size-Dependent
Toxicological Sciences·2016·182 cit.·DOI
8
Regulable neural progenitor-specific <i>Tsc1</i> loss yields giant cells with organellar dysfunction in a model of tuberous sclerosis complex
Proceedings of the National Academy of Sciences·2011·178 cit.·DOI
9
Tuberous Sclerosis Complex Activity Is Required to Control Neuronal Stress Responses in an mTOR-Dependent Manner
Journal of Neuroscience·2009·161 cit.·DOI
10
Impaired Autophagy in Neurons after Disinhibition of Mammalian Target of Rapamycin and Its Contribution to Epileptogenesis
Journal of Neuroscience·2012·152 cit.·DOI

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