Cell Signaling Technology

HDAC2 (3F3) Mouse Monoclonal Antibody

#5113 · western blot · $148,348

66

Evidence Score · Well-Established

Used by 313 independent labs across 344 papers. How we score this →

100

Citing papers

10.7k

Combined citations

Supplier page Find this product

Research areas

Autophagy in Disease and Therapy9 Cancer-related Molecular Pathways9 Epigenetics and DNA Methylation7 Ubiquitin and proteasome pathways7 MicroRNA in disease regulation6 DNA Repair Mechanisms5 Cancer-related molecular mechanisms research5 Cancer, Hypoxia, and Metabolism4

Papers citing this product

1

Papain-like protease regulates SARS-CoV-2 viral spread and innate immunity

Nature·2020·1.2k cit.·DOI

2

RETRACTED: Tanshinone I attenuates the malignant biological properties of ovarian cancer by inducing apoptosis and autophagy via the inactivation of PI3K/AKT/mTOR pathway

Cell Proliferation·2019·351 cit.·DOI

3

Particulate matter 2.5 damages skin cells by inducing oxidative stress, subcellular organelle dysfunction, and apoptosis

Archives of Toxicology·2018·331 cit.·DOI

4

Identification of a Novel Extracellular Cation-sensing G-protein-coupled Receptor

Journal of Biological Chemistry·2005·313 cit.·DOI

5

D-2-hydroxyglutarate produced by mutant IDH1 perturbs collagen maturation and basement membrane function

Genes & Development·2012·296 cit.·DOI

6

miR-142-5p and miR-130a-3p are regulated by IL-4 and IL-13 and control profibrogenic macrophage program

Nature Communications·2015·261 cit.·DOI

7

Proinvasive Properties of Ovarian Cancer Ascites-Derived Membrane Vesicles

Cancer Research·2004·246 cit.·DOI

8

Acetylation of MORC2 by NAT10 regulates cell-cycle checkpoint control and resistance to DNA-damaging chemotherapy and radiotherapy in breast cancer

Nucleic Acids Research·2020·228 cit.·DOI

9

IL10-driven STAT3 signalling in senescent macrophages promotes pathological eye angiogenesis

Nature Communications·2015·211 cit.·DOI

10

Aryl hydrocarbon receptor negatively regulates NLRP3 inflammasome activity by inhibiting NLRP3 transcription

Nature Communications·2014·204 cit.·DOI

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