Cell Signaling Technology

ISG15 (22D2) Rabbit Monoclonal Antibody

#2758 · $149

100
Citing papers
15k
Combined citations
Supplier page Find this product

Research areas

Sirtuins and Resveratrol in Medicine19Adipose Tissue and Metabolism12Autophagy in Disease and Therapy8Epigenetics and DNA Methylation5Protein Degradation and Inhibitors4Genomics, phytochemicals, and oxidative stress4Cancer-related Molecular Pathways4Neurogenesis and neuroplasticity mechanisms4

Papers citing this product

1
Sirt3 blocks the cardiac hypertrophic response by augmenting Foxo3a-dependent antioxidant defense mechanisms in mice
Journal of Clinical Investigation·2009·982 cit.·DOI
2
Accelerated MDM2 auto‐degradation induced by DNA‐damage kinases is required for p53 activation
The EMBO Journal·2004·390 cit.·DOI
3
Artemisinins Target GABAA Receptor Signaling and Impair α Cell Identity
Cell·2016·376 cit.·DOI
4
B cells sustain inflammation and predict response to immune checkpoint blockade in human melanoma
Nature Communications·2019·374 cit.·DOI
5
Efferocytosis induces a novel SLC program to promote glucose uptake and lactate release
Nature·2018·358 cit.·DOI
6
Iterative Design and Optimization of Initially Inactive Proteolysis Targeting Chimeras (PROTACs) Identify VZ185 as a Potent, Fast, and Selective von Hippel–Lindau (VHL) Based Dual Degrader Probe of BRD9 and BRD7
Journal of Medicinal Chemistry·2018·330 cit.·DOI
7
Silver nanoparticles have lethal and sublethal adverse effects on development and longevity by inducing ROS-mediated stress responses
Scientific Reports·2018·316 cit.·DOI
8
Syndecan-1 is a novel molecular marker for triple negative inflammatory breast cancer and modulates the cancer stem cell phenotype via the IL-6/STAT3, Notch and EGFR signaling pathways
Molecular Cancer·2017·276 cit.·DOI
9
Early glycolytic reprogramming controls microglial inflammatory activation
Journal of Neuroinflammation·2021·254 cit.·DOI
10
Omeprazole Blocks STAT6 Binding to the Eotaxin-3 Promoter in Eosinophilic Esophagitis Cells
PLoS ONE·2012·250 cit.·DOI

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