Cell Signaling Technology

FRMD6 (D8X3R) Rabbit Monoclonal Antibody

#14688 · $161

35
Citing papers
1k
Combined citations
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Research areas

Epigenetics and DNA Methylation4Cellular Mechanics and Interactions3Fungal and yeast genetics research3Autophagy in Disease and Therapy2Advanced Glycation End Products research2Biofuel production and bioconversion2Anesthesia and Neurotoxicity Research2Cancer-related molecular mechanisms research2

Papers citing this product

1
Fluid shear triggers microvilli formation via mechanosensitive activation of TRPV6
Nature Communications·2015·183 cit.·DOI
2
Diverse Regulation of the CreA Carbon Catabolite Repressor in <i>Aspergillus nidulans</i>
Genetics·2016·168 cit.·DOI
3
Akkermansia muciniphila and its metabolite propionic acid maintains neuronal mitochondrial division and autophagy homeostasis during Alzheimer’s disease pathologic process via GPR41 and GPR43
Microbiome·2025·72 cit.·DOI
4
FGF21 Attenuated LPS-Induced Depressive-Like Behavior via Inhibiting the Inflammatory Pathway
Frontiers in Pharmacology·2020·67 cit.·DOI
5
Disruption of zinc finger DNA binding domain in catabolite repressor Mig1 increases growth rate, hyphal branching, and cellulase expression in hypercellulolytic fungus Penicillium funiculosum NCIM1228
Biotechnology for Biofuels·2018·60 cit.·DOI
6
Mixed Linkage β-1,3/1,4-Glucan Oligosaccharides Induce Defense Responses in Hordeum vulgare and Arabidopsis thaliana
Frontiers in Plant Science·2021·56 cit.·DOI
7
Homozygous MTAP deletion in primary human glioblastoma is not associated with elevation of methylthioadenosine
Nature Communications·2021·53 cit.·DOI
8
Targeting EGFR-dependent tumors by disrupting an ARF6-mediated sorting system
Nature Communications·2022·50 cit.·DOI
9
Baicalein Attenuates Pyroptosis and Endoplasmic Reticulum Stress Following Spinal Cord Ischemia-Reperfusion Injury via Autophagy Enhancement
Frontiers in Pharmacology·2020·48 cit.·DOI
10
CRB3 regulates contact inhibition by activating the Hippo pathway in mammary epithelial cells
Cell Death and Disease·2017·47 cit.·DOI