Cell Signaling Technology

Argonaute 4 (D10F10) Rabbit Monoclonal Antibody

#6913 · $161

100

Citing papers

8.7k

Combined citations

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Research areas

PI3K/AKT/mTOR signaling in cancer16 Lung Cancer Treatments and Mutations9 Bone Metabolism and Diseases8 Endoplasmic Reticulum Stress and Disease7 Cancer-related Molecular Pathways7 Adipokines, Inflammation, and Metabolic Diseases6 Cell death mechanisms and regulation6 Bone health and treatments5

Papers citing this product

1

Autophagy inhibition enhances therapy-induced apoptosis in a Myc-induced model of lymphoma

Journal of Clinical Investigation·2007·1.1k cit.·DOI

2

Insulin resistance causes inflammation in adipose tissue

Journal of Clinical Investigation·2018·464 cit.·DOI

3

Lung cancers with acquired resistance to EGFR inhibitors occasionally harbor <i>BRAF</i> gene mutations but lack mutations in <i>KRAS, NRAS,</i> or <i>MEK1</i>

Proceedings of the National Academy of Sciences·2012·436 cit.·DOI

4

Divergent effects of glucose and fructose on hepatic lipogenesis and insulin signaling

Journal of Clinical Investigation·2017·340 cit.·DOI

5

Temporally sequenced anticancer drugs overcome adaptive resistance by targeting a vulnerable chemotherapy-induced phenotypic transition

Nature Communications·2015·286 cit.·DOI

6

Bone-specific insulin resistance disrupts whole-body glucose homeostasis via decreased osteocalcin activation

Journal of Clinical Investigation·2014·242 cit.·DOI

7

Sonic Hedgehog Pathway Activation Is Induced by Acute Brain Injury and Regulated by Injury-Related Inflammation

Journal of Neuroscience·2009·199 cit.·DOI

8

The Notch ligand Jagged2 promotes lung adenocarcinoma metastasis through a miR-200–dependent pathway in mice

Journal of Clinical Investigation·2011·197 cit.·DOI

9

Osteoclast Response to Low Extracellular Sodium and the Mechanism of Hyponatremia-induced Bone Loss

Journal of Biological Chemistry·2010·188 cit.·DOI

10

Convergent Akt activation drives acquired EGFR inhibitor resistance in lung cancer

Nature Communications·2017·182 cit.·DOI