Cell Signaling Technology

Mono-Methyl-Histone H3 (Lys4) (D1A9) Rabbit Monoclonal Antibody

#5326 · $179

100
Citing papers
9.1k
Combined citations
Supplier page Find this product

Research areas

Cytokine Signaling Pathways and Interactions12Cancer Cells and Metastasis10Epigenetics and DNA Methylation9Cancer-related molecular mechanisms research8Wnt/β-catenin signaling in development and cancer8Protein Kinase Regulation and GTPase Signaling7RNA modifications and cancer7PI3K/AKT/mTOR signaling in cancer7

Papers citing this product

1
Receptor interacting protein kinase 2–mediated mitophagy regulates inflammasome activation during virus infection
Nature Immunology·2013·383 cit.·DOI
2
Hepatitis C virus-induced up-regulation of microRNA-155 promotes hepatocarcinogenesis by activating Wnt signaling
Hepatology·2012·300 cit.·DOI
3
<i>miR-146a</i> controls the resolution of T cell responses in mice
The Journal of Experimental Medicine·2012·277 cit.·DOI
4
TCR ligand density and affinity determine peripheral induction of Foxp3 in vivo
The Journal of Experimental Medicine·2010·271 cit.·DOI
5
Organogenesis relies on SoxC transcription factors for the survival of neural and mesenchymal progenitors
Nature Communications·2010·229 cit.·DOI
6
A Th17‐like developmental process leads to CD8<sup>+</sup> Tc17 cells with reduced cytotoxic activity
European Journal of Immunology·2009·223 cit.·DOI
7
BET bromodomain inhibition enhances T cell persistence and function in adoptive immunotherapy models
Journal of Clinical Investigation·2016·214 cit.·DOI
8
CKAP4 is a Dickkopf1 receptor and is involved in tumor progression
Journal of Clinical Investigation·2016·182 cit.·DOI
9
Thymoquinone inhibits proliferation, induces apoptosis and chemosensitizes human multiple myeloma cells through suppression of signal transducer and activator of transcription 3 activation pathway
British Journal of Pharmacology·2010·174 cit.·DOI
10
SOX2 Gene Regulates the Transcriptional Network of Oncogenes and Affects Tumorigenesis of Human Lung Cancer Cells
PLoS ONE·2012·163 cit.·DOI

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