Cell Signaling Technology

Phospho-Akt Substrate (RXXS*/T*) (110B7E) Rabbit Monoclonal Antibody

#9614 · $168

100
Citing papers
7.1k
Combined citations
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Research areas

PI3K/AKT/mTOR signaling in cancer10Epigenetics and DNA Methylation9Cell death mechanisms and regulation7RNA Research and Splicing7RNA modifications and cancer6Autophagy in Disease and Therapy6Estrogen and related hormone effects6Cancer-related molecular mechanisms research5

Papers citing this product

1
PTEN Loss Confers BRAF Inhibitor Resistance to Melanoma Cells through the Suppression of BIM Expression
Cancer Research·2011·546 cit.·DOI
2
Cell-cycle-regulated activation of Akt kinase by phosphorylation at its carboxyl terminus
Nature·2014·366 cit.·DOI
3
Possible Contributions of Reactive Oxygen Species and Mitogen-Activated Protein Kinase to Renal Injury in Aldosterone/Salt-Induced Hypertensive Rats
Hypertension·2004·310 cit.·DOI
4
The 16p11.2 Deletion Mouse Model of Autism Exhibits Altered Cortical Progenitor Proliferation and Brain Cytoarchitecture Linked to the ERK MAPK Pathway
Journal of Neuroscience·2015·222 cit.·DOI
5
Flexible patch with printable and antibacterial conductive hydrogel electrodes for accelerated wound healing
Biomaterials·2022·198 cit.·DOI
6
MiR-31 promotes mammary stem cell expansion and breast tumorigenesis by suppressing Wnt signaling antagonists
Nature Communications·2017·197 cit.·DOI
7
Exosomal microRNA miR-1246 induces cell motility and invasion through the regulation of DENND2D in oral squamous cell carcinoma
Scientific Reports·2016·195 cit.·DOI
8
The ERK MAPK Pathway Is Essential for Skeletal Development and Homeostasis
International Journal of Molecular Sciences·2019·191 cit.·DOI
9
Chronic Estradiol Administration In Vivo Promotes the Proinflammatory Response of Macrophages to TLR4 Activation: Involvement of the Phosphatidylinositol 3-Kinase Pathway
The Journal of Immunology·2008·182 cit.·DOI
10
The LKB1-salt-inducible kinase pathway functions as a key gluconeogenic suppressor in the liver
Nature Communications·2014·163 cit.·DOI

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