Cell Signaling Technology

Phospho-CDCP1 (Tyr743) (D2G2J) Rabbit Monoclonal Antibody

#14965 · $176

100
Citing papers
2.6k
Combined citations
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Research areas

Genomics, phytochemicals, and oxidative stress24Glutathione Transferases and Polymorphisms10Liver Disease Diagnosis and Treatment7Neuroinflammation and Neurodegeneration Mechanisms5interferon and immune responses4Cancer Cells and Metastasis4Bioactive Compounds and Antitumor Agents4Heme Oxygenase-1 and Carbon Monoxide4

Papers citing this product

1
Placental Mesenchymal Stem Cell-Derived Extracellular Vesicles Promote Myelin Regeneration in an Animal Model of Multiple Sclerosis
Cells·2019·138 cit.·DOI
2
NRF2 Activation Restores Disease Related Metabolic Deficiencies in Olfactory Neurosphere-Derived Cells from Patients with Sporadic Parkinson's Disease
PLoS ONE·2011·138 cit.·DOI
3
Wilms Tumor Gene on X Chromosome (WTX) Inhibits Degradation of NRF2 Protein through Competitive Binding to KEAP1 Protein
Journal of Biological Chemistry·2012·132 cit.·DOI
4
Adenomatous Polyposis Coli Regulates Oligodendroglial Development
Journal of Neuroscience·2013·106 cit.·DOI
5
Zeaxanthin induces Nrf2-mediated phase II enzymes in protection of cell death
Cell Death and Disease·2014·102 cit.·DOI
6
ERK2 regulates epithelial-to-mesenchymal plasticity through DOCK10-dependent Rac1/FoxO1 activation
Proceedings of the National Academy of Sciences·2019·97 cit.·DOI
7
Keap1 Cysteine 288 as a Potential Target for Diallyl Trisulfide-Induced Nrf2 Activation
PLoS ONE·2014·90 cit.·DOI
8
Selective Reconstitution and Recovery of Functional γ-Secretase Complex on Budded Baculovirus Particles
Journal of Biological Chemistry·2004·81 cit.·DOI
9
The Inflammasome Pyrin Contributes to Pertussis Toxin-Induced IL-1β Synthesis, Neutrophil Intravascular Crawling and Autoimmune Encephalomyelitis
PLoS Pathogens·2014·80 cit.·DOI
10
NRF2-driven miR-125B1 and miR-29B1 transcriptional regulation controls a novel anti-apoptotic miRNA regulatory network for AML survival
Cell Death and Differentiation·2014·68 cit.·DOI