Cell Signaling Technology

Tyrosine Hydroxylase (E2L6M) Rabbit Monoclonal Antibody (Alexa Fluor® 647 Conjugate)

#80331 · $465

13
Citing papers
149
Combined citations
Supplier page Find this product

Research areas

Developmental Biology and Gene Regulation3Congenital heart defects research2Autophagy in Disease and Therapy1Bioactive Compounds and Antitumor Agents1Cancer-related molecular mechanisms research1Cellular Mechanics and Interactions1Chromatography in Natural Products1Axon Guidance and Neuronal Signaling1

Papers citing this product

1
Isoaaptamine Induces T-47D Cells Apoptosis and Autophagy via Oxidative Stress
Marine Drugs·2018·45 cit.·DOI
2
Presenilin/γ-secretase-dependent EphA3 processing mediates axon elongation through non-muscle myosin IIA
eLife·2019·20 cit.·DOI
3
Calcium Signaling and Mitochondrial Function in Presenilin 2 Knock-Out Mice: Looking for Any Loss-of-Function Phenotype Related to Alzheimer’s Disease
Cells·2021·18 cit.·DOI
4
Actin crosslinking by α-actinin averts viscous dissipation of myosin force transmission in stress fibers
iScience·2023·17 cit.·DOI
5
Regulation of Nodal signaling propagation by receptor interactions and positive feedback
eLife·2022·16 cit.·DOI
6
AP-2 Adaptor Complex-Dependent Enhancement of HIV-1 Replication by Nef in the Absence of the Nef/AP-2 Targets SERINC5 and CD4
mBio·2023·6 cit.·DOI
7
Panaxadiol Attenuates Neuronal Oxidative Stress and Apoptosis in Cerebral Ischemia/Reperfusion Injury via Regulation of the <scp>JAK3</scp> / <scp>STAT3</scp> / <scp>HIF</scp> ‐1α Signaling Pathway
CNS Neuroscience & Therapeutics·2025·6 cit.·DOI
8
Dicer-dependent production of microRNA221 in hepatocytes inhibits p27 and is required for liver regeneration in mice
American Journal of Physiology-Gastrointestinal and Liver Physiology·2017·6 cit.·DOI
9
The pattern of Nodal morphogen signaling is shaped by co-receptor expression
bioRxiv (Cold Spring Harbor Laboratory)·2019·5 cit.·DOI
10
Extensive crosstalk of G protein-coupled receptors with the Hedgehog signalling pathway
Development·2021·4 cit.·DOI

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