Cell Signaling Technology

Phospho-LAT (Tyr171) Antibody

#3581 · $362

100
Citing papers
10.5k
Combined citations
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Research areas

Neurogenesis and neuroplasticity mechanisms8Immune Cell Function and Interaction7Nerve injury and regeneration7Neuroinflammation and Neurodegeneration Mechanisms6MicroRNA in disease regulation5Endoplasmic Reticulum Stress and Disease5Immunotherapy and Immune Responses5Mesenchymal stem cell research5

Papers citing this product

1
Cartilage Tissue Engineering by the 3D Bioprinting of iPS Cells in a Nanocellulose/Alginate Bioink
Scientific Reports·2017·475 cit.·DOI
2
Fibrinogen Triggers Astrocyte Scar Formation by Promoting the Availability of Active TGF-β after Vascular Damage
Journal of Neuroscience·2010·374 cit.·DOI
3
Recruitment of folliculin to lysosomes supports the amino acid–dependent activation of Rag GTPases
The Journal of Cell Biology·2013·351 cit.·DOI
4
A substrate-specific mTORC1 pathway underlies Birt–Hogg–Dubé syndrome
Nature·2020·315 cit.·DOI
5
MicroRNA-99a Inhibits Hepatocellular Carcinoma Growth and Correlates with Prognosis of Patients with Hepatocellular Carcinoma
Journal of Biological Chemistry·2011·248 cit.·DOI
6
The Protective Role of Smad7 in Diabetic Kidney Disease: Mechanism and Therapeutic Potential
Diabetes·2010·224 cit.·DOI
7
Regulation of Global Acetylation in Mitosis through Loss of Histone Acetyltransferases and Deacetylases from Chromatin
Journal of Biological Chemistry·2001·211 cit.·DOI
8
Inhibition of Classic Signaling Is a Novel Function of Soluble Glycoprotein 130 (sgp130), Which Is Controlled by the Ratio of Interleukin 6 and Soluble Interleukin 6 Receptor
Journal of Biological Chemistry·2011·206 cit.·DOI
9
Mesenchymal Stem Cells Transmigrate Between and Directly Through Tumor Necrosis Factor‐α‐Activated Endothelial Cells Via Both Leukocyte‐Like and Novel Mechanisms
Stem Cells·2012·196 cit.·DOI
10
CCL2 Mediates Neuron–Macrophage Interactions to Drive Proregenerative Macrophage Activation Following Preconditioning Injury
Journal of Neuroscience·2015·189 cit.·DOI

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