Cell Signaling Technology

alpha-Amylase Antibody

#4017 · $317

100
Citing papers
17.8k
Combined citations
Supplier page Find this product

Research areas

Pain Mechanisms and Treatments28Nerve injury and regeneration16Neuroinflammation and Neurodegeneration Mechanisms7Alzheimer's disease research and treatments6Pancreatic function and diabetes5Endoplasmic Reticulum Stress and Disease5Neuropeptides and Animal Physiology5Cell death mechanisms and regulation4

Papers citing this product

1
Glycosylation and stabilization of programmed death ligand-1 suppresses T-cell activity
Nature Communications·2016·1.1k cit.·DOI
2
Human iPSC-Derived Natural Killer Cells Engineered with Chimeric Antigen Receptors Enhance Anti-tumor Activity
Cell stem cell·2018·980 cit.·DOI
3
Activated Microglia Contribute to the Maintenance of Chronic Pain after Spinal Cord Injury
Journal of Neuroscience·2006·633 cit.·DOI
4
TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury
Journal of Clinical Investigation·2005·612 cit.·DOI
5
Maternal high-fat diet triggers lipotoxicity in the fetal livers of nonhuman primates
Journal of Clinical Investigation·2009·593 cit.·DOI
6
JNK-Induced MCP-1 Production in Spinal Cord Astrocytes Contributes to Central Sensitization and Neuropathic Pain
Journal of Neuroscience·2009·564 cit.·DOI
7
A Peptide c-Jun N-Terminal Kinase (JNK) Inhibitor Blocks Mechanical Allodynia after Spinal Nerve Ligation: Respective Roles of JNK Activation in Primary Sensory Neurons and Spinal Astrocytes for Neuropathic Pain Development and Maintenance
Journal of Neuroscience·2006·519 cit.·DOI
8
Acute p38-Mediated Modulation of Tetrodotoxin-Resistant Sodium Channels in Mouse Sensory Neurons by Tumor Necrosis Factor-α
Journal of Neuroscience·2006·484 cit.·DOI
9
P2X4-Receptor-Mediated Synthesis and Release of Brain-Derived Neurotrophic Factor in Microglia Is Dependent on Calcium and p38-Mitogen-Activated Protein Kinase Activation
Journal of Neuroscience·2009·463 cit.·DOI
10
Scutellarin ameliorates pulmonary fibrosis through inhibiting NF-κB/NLRP3-mediated epithelial–mesenchymal transition and inflammation
Cell Death and Disease·2020·387 cit.·DOI

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