Cell Signaling Technology

Phospho-c-Myc (Ser62) (E1J4K) Rabbit Monoclonal Antibody

#13748 · $197

100
Citing papers
3k
Combined citations
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Research areas

Cancer, Hypoxia, and Metabolism15PARP inhibition in cancer therapy10RNA modifications and cancer8Cancer-related Molecular Pathways8Metabolism, Diabetes, and Cancer6Cancer, Lipids, and Metabolism6Cell death mechanisms and regulation6Cancer Cells and Metastasis5

Papers citing this product

1
PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stress
Cell Death and Differentiation·2012·832 cit.·DOI
2
Integrin CD11b activation drives anti-tumor innate immunity
Nature Communications·2018·313 cit.·DOI
3
Inhibition of H1N1 influenza virus-induced apoptosis by functionalized selenium nanoparticles with amantadine through ROS-mediated AKT signaling pathways
International Journal of Nanomedicine·2018·132 cit.·DOI
4
Coronavirus Infection Induces DNA Replication Stress Partly through Interaction of Its Nonstructural Protein 13 with the p125 Subunit of DNA Polymerase δ
Journal of Biological Chemistry·2011·100 cit.·DOI
5
DOX-loaded pH-sensitive mesoporous silica nanoparticles coated with PDA and PEG induce pro-death autophagy in breast cancer
RSC Advances·2017·93 cit.·DOI
6
Hyperbaric Oxygen Reduces Infarction Volume and Hemorrhagic Transformation Through ATP/NAD <sup>+</sup> /Sirt1 Pathway in Hyperglycemic Middle Cerebral Artery Occlusion Rats
Stroke·2017·87 cit.·DOI
7
Latency-Reversing Agents Induce Differential Responses in Distinct Memory CD4 T Cell Subsets in Individuals on Antiretroviral Therapy
Cell Reports·2019·64 cit.·DOI
8
Modulation of Inflammasome and Pyroptosis by Olaparib, a PARP-1 Inhibitor, in the R6/2 Mouse Model of Huntington’s Disease
Cells·2020·64 cit.·DOI
9
Towards Resolving the Pro- and Anti-Tumor Effects of the Aryl Hydrocarbon Receptor
International Journal of Molecular Sciences·2018·62 cit.·DOI
10
Irreversible cell cycle exit associated with senescence is mediated by constitutive MYC degradation
Cell Reports·2023·51 cit.·DOI

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