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Cell Signaling Technology
PathScan® Phospho-GSK-3 beta (Ser9) Sandwich ELISA Kit
#7311 · $661
100
Citing papers
14.9k
Combined citations
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Research areas
Immune cells in cancer
23
MicroRNA in disease regulation
8
Phagocytosis and Immune Regulation
8
Immune Cell Function and Interaction
8
Adipose Tissue and Metabolism
7
Stress Responses and Cortisol
7
Epigenetics and DNA Methylation
6
CAR-T cell therapy research
6
Papers citing this product
1
A New Mesenchymal Stem Cell (MSC) Paradigm: Polarization into a Pro-Inflammatory MSC1 or an Immunosuppressive MSC2 Phenotype
PLoS ONE
·
2010
·
1.2k cit.
·
DOI
2
Reactive Oxygen Species as a Signal in Glucose-Stimulated Insulin Secretion
Diabetes
·
2007
·
569 cit.
·
DOI
3
TGF-β induces M2-like macrophage polarization via SNAIL-mediated suppression of a pro-inflammatory phenotype
Oncotarget
·
2016
·
564 cit.
·
DOI
4
Inhibition of Monocyte Adhesion to Endothelial Cells and Attenuation of Atherosclerotic Lesion by a Glucagon-like Peptide-1 Receptor Agonist, Exendin-4
Diabetes
·
2010
·
533 cit.
·
DOI
5
The short-chain fatty acid pentanoate suppresses autoimmunity by modulating the metabolic-epigenetic crosstalk in lymphocytes
Nature Communications
·
2019
·
509 cit.
·
DOI
6
Epithelial NOTCH Signaling Rewires the Tumor Microenvironment of Colorectal Cancer to Drive Poor-Prognosis Subtypes and Metastasis
Cancer Cell
·
2019
·
441 cit.
·
DOI
7
Mood Stabilizer Valproate Promotes ERK Pathway-Dependent Cortical Neuronal Growth and Neurogenesis
Journal of Neuroscience
·
2004
·
404 cit.
·
DOI
8
Monocyte and M1 Macrophage-induced Barrier Defect Contributes to Chronic Intestinal Inflammation in IBD
Inflammatory Bowel Diseases
·
2015
·
361 cit.
·
DOI
9
Nucleoporin NUP153 Phenylalanine-Glycine Motifs Engage a Common Binding Pocket within the HIV-1 Capsid Protein to Mediate Lentiviral Infectivity
PLoS Pathogens
·
2013
·
294 cit.
·
DOI
10
Blocking immunoinhibitory receptor LILRB2 reprograms tumor-associated myeloid cells and promotes antitumor immunity
Journal of Clinical Investigation
·
2018
·
262 cit.
·
DOI
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