Cell Signaling Technology

MIB1 Antibody

#4400 · $317

60
Evidence Score · Well-Established
Used by 91 independent labs across 100 papers. How we score this →
100
Citing papers
12.7k
Combined citations
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Research areas

Estrogen and related hormone effects8Immune Cell Function and Interaction8Epigenetics and DNA Methylation7RNA modifications and cancer6Menopause: Health Impacts and Treatments5Cancer-related molecular mechanisms research5Angiogenesis and VEGF in Cancer4Cancer-related Molecular Pathways4

Papers citing this product

1
AZD2171: A Highly Potent, Orally Bioavailable, Vascular Endothelial Growth Factor Receptor-2 Tyrosine Kinase Inhibitor for the Treatment of Cancer
Cancer Research·2005·710 cit.·DOI
2
The dynamic interaction of AMBRA1 with the dynein motor complex regulates mammalian autophagy
The Journal of Cell Biology·2010·488 cit.·DOI
3
Preclinical characterization of anlotinib, a highly potent and selective vascular endothelial growth factor receptor‐2 inhibitor
Cancer Science·2018·368 cit.·DOI
4
Reversible inhibitor of p97, DBeQ, impairs both ubiquitin-dependent and autophagic protein clearance pathways
Proceedings of the National Academy of Sciences·2011·323 cit.·DOI
5
Orchestration of lymphocyte chemotaxis by mitochondrial dynamics
The Journal of Experimental Medicine·2006·321 cit.·DOI
6
Predicting clinical response to anticancer drugs using an ex vivo platform that captures tumour heterogeneity
Nature Communications·2015·307 cit.·DOI
7
Minocycline Alleviates Death of Oligodendrocytes by Inhibiting Pro-Nerve Growth Factor Production in Microglia after Spinal Cord Injury
Journal of Neuroscience·2007·270 cit.·DOI
8
Transcription factor EGR1 directs tendon differentiation and promotes tendon repair
Journal of Clinical Investigation·2013·262 cit.·DOI
9
Long noncoding RNA AGPG regulates PFKFB3-mediated tumor glycolytic reprogramming
Nature Communications·2020·258 cit.·DOI
10
Inflammation mobilizes copper metabolism to promote colon tumorigenesis via an IL-17-STEAP4-XIAP axis
Nature Communications·2020·239 cit.·DOI

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