Cell Signaling Technology

PathScan® Phospho-TrkA (Tyr490) Sandwich ELISA Kit

#7210 · $661

100
Citing papers
7.1k
Combined citations
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Research areas

Autophagy in Disease and Therapy8MicroRNA in disease regulation6Mitochondrial Function and Pathology6Immune Response and Inflammation5Cancer, Hypoxia, and Metabolism5Inflammasome and immune disorders5Gut microbiota and health4Endoplasmic Reticulum Stress and Disease4

Papers citing this product

1
The eIF2α/ATF4 pathway is essential for stress-induced autophagy gene expression
Nucleic Acids Research·2013·1.1k cit.·DOI
2
Melanoma addiction to the long non-coding RNA SAMMSON
Nature·2016·588 cit.·DOI
3
Intravenous immunoglobulin suppresses NLRP1 and NLRP3 inflammasome-mediated neuronal death in ischemic stroke
Cell Death and Disease·2013·490 cit.·DOI
4
N1-methyladenosine methylation in tRNA drives liver tumourigenesis by regulating cholesterol metabolism
Nature Communications·2021·253 cit.·DOI
5
Nimbolide protects against endotoxin-induced acute respiratory distress syndrome by inhibiting TNF-α mediated NF-κB and HDAC-3 nuclear translocation
Cell Death and Disease·2019·148 cit.·DOI
6
Piperlongumine regulates epigenetic modulation and alleviates psoriasis-like skin inflammation via inhibition of hyperproliferation and inflammation
Cell Death and Disease·2020·145 cit.·DOI
7
Phosphatidic Acid Regulates Systemic Inflammatory Responses by Modulating the Akt-Mammalian Target of Rapamycin-p70 S6 Kinase 1 Pathway
Journal of Biological Chemistry·2003·142 cit.·DOI
8
The endocannabinoid system and pivotal role of the CB <sub>2</sub> receptor in mouse spermatogenesis
Proceedings of the National Academy of Sciences·2009·136 cit.·DOI
9
Oxidative and pro-inflammatory impact of regular and denicotinized cigarettes on blood brain barrier endothelial cells: is smoking reduced or nicotine-free products really safe?
BMC Neuroscience·2014·125 cit.·DOI
10
Phosphorylation status determines the opposing functions of Smad2/Smad3 as STAT3 cofactors in TH17 differentiation
Nature Communications·2015·124 cit.·DOI