Cell Signaling Technology

FRA1 (F2C9L) Rabbit Monoclonal Antibody (IHC Formulated)

#28801 · $189

37
Citing papers
900
Combined citations
Supplier page Find this product

Research areas

Cancer, Hypoxia, and Metabolism5Autophagy in Disease and Therapy5Melanoma and MAPK Pathways4RNA modifications and cancer3Alzheimer's disease research and treatments3Circular RNAs in diseases3Cancer-related molecular mechanisms research3Cancer Cells and Metastasis2

Papers citing this product

1
Insulin-degrading enzyme secretion from astrocytes is mediated by an autophagy-based unconventional secretory pathway in Alzheimer disease
Autophagy·2016·129 cit.·DOI
2
Impaired neutrophil activity and increased susceptibility to bacterial infection in mice lacking glucose-6-phosphatase–β
Journal of Clinical Investigation·2007·121 cit.·DOI
3
Periostin is identified as a putative metastatic marker in breast cancer-derived exosomes
Oncotarget·2016·68 cit.·DOI
4
Dendrobium nobile Lindl. Alkaloids Decreases the Level of Intracellular β-Amyloid by Improving Impaired Autolysosomal Proteolysis in APP/PS1 Mice
Frontiers in Pharmacology·2018·64 cit.·DOI
5
Retinoic Acid Suppresses the Canonical Wnt Signaling Pathway in Embryonic Stem Cells and Activates the Noncanonical Wnt Signaling Pathway
Stem Cells·2014·61 cit.·DOI
6
Evidence for Transcriptional Regulation of the Glucose-6-Phosphate Transporter by HIF-1<i>α</i>: Targeting G6PT with Mumbaistatin Analogs in Hypoxic Mesenchymal Stromal Cells
Stem Cells·2008·53 cit.·DOI
7
NFE2-Related Transcription Factor 2 Coordinates Antioxidant Defense with Thyroglobulin Production and Iodination in the Thyroid Gland
Thyroid·2018·52 cit.·DOI
8
Oncostatin M Is a Growth Factor for Ewing Sarcoma
American Journal Of Pathology·2012·42 cit.·DOI
9
Pseudoginsenoside‐F11 attenuates cerebral ischemic injury by alleviating autophagic/lysosomal defects
CNS Neuroscience & Therapeutics·2017·41 cit.·DOI
10
High miR-122 expression promotes malignant phenotypes in ccRCC by targeting occludin
International Journal of Oncology·2017·38 cit.·DOI

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