Cell Signaling Technology

Phospho-Vimentin (Ser56) (D5H2) Rabbit Monoclonal Antibody

#7391 · $176

100
Citing papers
12.5k
Combined citations
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Research areas

Cytokine Signaling Pathways and Interactions14PI3K/AKT/mTOR signaling in cancer10Cell death mechanisms and regulation9Cancer, Hypoxia, and Metabolism7Cancer Mechanisms and Therapy6Cancer-related Molecular Pathways6Histone Deacetylase Inhibitors Research6Inflammasome and immune disorders6

Papers citing this product

1
CD44 splice isoform switching in human and mouse epithelium is essential for epithelial-mesenchymal transition and breast cancer progression
Journal of Clinical Investigation·2011·647 cit.·DOI
2
Mutations causing syndromic autism define an axis of synaptic pathophysiology
Nature·2011·598 cit.·DOI
3
RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury
Cell Death and Differentiation·2016·439 cit.·DOI
4
Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress
Science·2013·410 cit.·DOI
5
Orally bioavailable small-molecule inhibitor of transcription factor Stat3 regresses human breast and lung cancer xenografts
Proceedings of the National Academy of Sciences·2012·347 cit.·DOI
6
Activation of STAT3 integrates common profibrotic pathways to promote fibroblast activation and tissue fibrosis
Nature Communications·2017·342 cit.·DOI
7
Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling
Proceedings of the National Academy of Sciences·2013·321 cit.·DOI
8
Amplification of PVT-1 is involved in poor prognosis via apoptosis inhibition in colorectal cancers
British Journal of Cancer·2013·288 cit.·DOI
9
Liver‐specific deletion of histone deacetylase 3 disrupts metabolic transcriptional networks
The EMBO Journal·2008·265 cit.·DOI
10
Stat3 signaling in acute myeloid leukemia: ligand-dependent and -independent activation and induction of apoptosis by a novel small-molecule Stat3 inhibitor
Blood·2011·261 cit.·DOI

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