Cell Signaling Technology

Rig-I (D14G6) Rabbit Monoclonal Antibody

#3743 · $149,348

68

Evidence Score · Well-Established

Used by 189 independent labs across 198 papers. How we score this →

100

Citing papers

11.1k

Combined citations

Supplier page Find this product

Research areas

interferon and immune responses11 Genomics, phytochemicals, and oxidative stress7 Immune Response and Inflammation6 NF-κB Signaling Pathways6 PI3K/AKT/mTOR signaling in cancer5 MicroRNA in disease regulation5 Mosquito-borne diseases and control5 Cancer, Hypoxia, and Metabolism4

Papers citing this product

1

Nrf2 and Keap1 Abnormalities in Non–Small Cell Lung Carcinoma and Association with Clinicopathologic Features

Clinical Cancer Research·2010·422 cit.·DOI

2

Coronavirus nonstructural protein 15 mediates evasion of dsRNA sensors and limits apoptosis in macrophages

Proceedings of the National Academy of Sciences·2017·407 cit.·DOI

3

Identification and characterization of pleckstrin-homology-domain-dependent and isoenzyme-specific Akt inhibitors

Biochemical Journal·2005·396 cit.·DOI

4

Glioblastomas acquire myeloid-affiliated transcriptional programs via epigenetic immunoediting to elicit immune evasion

Cell·2021·357 cit.·DOI

5

Transcription factor NRF2 regulates miR-1 and miR-206 to drive tumorigenesis

Journal of Clinical Investigation·2013·328 cit.·DOI

6

Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma

Molecular Cancer·2014·228 cit.·DOI

7

Akt activation and localisation correlate with tumour invasion and oncogene expression in thyroid cancer

Journal of Medical Genetics·2004·211 cit.·DOI

8

Quercetin inhibition of tumor invasion via suppressing PKC /ERK/AP-1-dependent matrix metalloproteinase-9 activation in breast carcinoma cells

Carcinogenesis·2008·209 cit.·DOI

9

Histone H3.3 maintains genome integrity during mammalian development

Genes & Development·2015·196 cit.·DOI

10

A genetic basis for the variation in the vulnerability of cancer to DNA damage

Nature Communications·2016·185 cit.·DOI

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