Cell Signaling Technology

Phospho-TGF-beta Receptor I (Ser165) (F5E2G) Rabbit Monoclonal Antibody

#26664 · $176,437

23

Citing papers

1k

Combined citations

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Research areas

Cell death mechanisms and regulation10 Ubiquitin and proteasome pathways7 Macrophage Migration Inhibitory Factor4 Cancer-related Molecular Pathways4 Asthma and respiratory diseases2 Helminth infection and control2 Autophagy in Disease and Therapy2 Endoplasmic Reticulum Stress and Disease2

Papers citing this product

1

Dual-targeting biomimetic delivery for anti-glioma activity<i>via</i>remodeling the tumor microenvironment and directing macrophage-mediated immunotherapy

Chemical Science·2018·248 cit.·DOI

2

The E3 Ubiquitin Ligase cIAP1 Binds and Ubiquitinates Caspase-3 and -7 via Unique Mechanisms at Distinct Steps in Their Processing

Journal of Biological Chemistry·2009·171 cit.·DOI

3

The Role of Macrophage Migration Inhibitory Factor in Autoimmune Liver Disease

Hepatology·2013·106 cit.·DOI

4

Livin promotes Smac/DIABLO degradation by ubiquitin–proteasome pathway

Cell Death and Differentiation·2006·90 cit.·DOI

5

Nuclear expression of survivin is associated with improved survival in metastatic ovarian carcinoma

Cancer·2006·71 cit.·DOI

6

Drosophila IAP1-Mediated Ubiquitylation Controls Activation of the Initiator Caspase DRONC Independent of Protein Degradation

PLoS Genetics·2011·56 cit.·DOI

7

Phosphorylation of Caspase-8 (Thr-263) by Ribosomal S6 Kinase 2 (RSK2) Mediates Caspase-8 Ubiquitination and Stability

Journal of Biological Chemistry·2010·52 cit.·DOI

8

cIAP1 Cooperatively Inhibits Procaspase-3 Activation by the Caspase-9 Apoptosome

Journal of Biological Chemistry·2010·34 cit.·DOI

9

JAK3 inhibitor suppresses multipotent ILC2s and attenuates steroid-resistant asthma

Science Advances·2023·31 cit.·DOI

10

Naringin protects H9C2 cardiomyocytes from chemical hypoxia‑induced injury by promoting the autophagic flux via the activation of the HIF‑1α/BNIP3 signaling pathway

International Journal of Molecular Medicine·2021·26 cit.·DOI

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