Cell Signaling Technology

Phospho-Chk1 (Ser317) Antibody

#2344 · $150,863

65

Evidence Score · Well-Established

Used by 283 independent labs across 302 papers. How we score this →

100

Citing papers

16.8k

Combined citations

Supplier page Find this product

Research areas

DNA Repair Mechanisms13 Cancer-related Molecular Pathways7 Wnt/β-catenin signaling in development and cancer7 Genomics and Chromatin Dynamics7 Estrogen and related hormone effects6 CRISPR and Genetic Engineering6 Ubiquitin and proteasome pathways6 Genetics and Neurodevelopmental Disorders5

Papers citing this product

1

Differential PROTAC substrate specificity dictated by orientation of recruited E3 ligase

Nature Communications·2019·516 cit.·DOI

2

Mood Stabilizer Valproate Promotes ERK Pathway-Dependent Cortical Neuronal Growth and Neurogenesis

Journal of Neuroscience·2004·404 cit.·DOI

3

MYC pathway activation in triple-negative breast cancer is synthetic lethal with CDK inhibition

The Journal of Experimental Medicine·2012·395 cit.·DOI

4

PPM1D dephosphorylates Chk1 and p53 and abrogates cell cycle checkpoints

Genes & Development·2005·383 cit.·DOI

5

Gli1 identifies osteogenic progenitors for bone formation and fracture repair

Nature Communications·2017·353 cit.·DOI

6

Behavioral improvement in a primate Parkinson's model is associated with multiple homeostatic effects of human neural stem cells

Proceedings of the National Academy of Sciences·2007·344 cit.·DOI

7

BRCA locus-specific loss of heterozygosity in germline BRCA1 and BRCA2 carriers

Nature Communications·2017·308 cit.·DOI

8

Neuroprotective Effects of Regulators of the Glycogen Synthase Kinase-3β Signaling Pathway in a Transgenic Model of Alzheimer's Disease Are Associated with Reduced Amyloid Precursor Protein Phosphorylation

Journal of Neuroscience·2007·287 cit.·DOI

9

Expression of the Long Non-Coding RNA HOTAIR Correlates with Disease Progression in Bladder Cancer and Is Contained in Bladder Cancer Patient Urinary Exosomes

PLoS ONE·2016·274 cit.·DOI

10

Senescence-associated heterochromatin foci are dispensable for cellular senescence, occur in a cell type- and insult-dependent manner and follow expression of p16<sup>ink4a</sup>

Cell Cycle·2011·269 cit.·DOI

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