Cell Signaling Technology

Phospho-Chk1 (Ser317) Antibody

#2344 · $150

100
Citing papers
16.8k
Combined citations
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Research areas

DNA Repair Mechanisms13Cancer-related Molecular Pathways7Wnt/β-catenin signaling in development and cancer7Genomics and Chromatin Dynamics7Estrogen and related hormone effects6CRISPR and Genetic Engineering6Ubiquitin and proteasome pathways6Genetics and Neurodevelopmental Disorders5

Papers citing this product

1
Differential PROTAC substrate specificity dictated by orientation of recruited E3 ligase
Nature Communications·2019·516 cit.·DOI
2
Mood Stabilizer Valproate Promotes ERK Pathway-Dependent Cortical Neuronal Growth and Neurogenesis
Journal of Neuroscience·2004·404 cit.·DOI
3
MYC pathway activation in triple-negative breast cancer is synthetic lethal with CDK inhibition
The Journal of Experimental Medicine·2012·395 cit.·DOI
4
PPM1D dephosphorylates Chk1 and p53 and abrogates cell cycle checkpoints
Genes & Development·2005·383 cit.·DOI
5
Gli1 identifies osteogenic progenitors for bone formation and fracture repair
Nature Communications·2017·353 cit.·DOI
6
Behavioral improvement in a primate Parkinson's model is associated with multiple homeostatic effects of human neural stem cells
Proceedings of the National Academy of Sciences·2007·344 cit.·DOI
7
BRCA locus-specific loss of heterozygosity in germline BRCA1 and BRCA2 carriers
Nature Communications·2017·308 cit.·DOI
8
Neuroprotective Effects of Regulators of the Glycogen Synthase Kinase-3β Signaling Pathway in a Transgenic Model of Alzheimer's Disease Are Associated with Reduced Amyloid Precursor Protein Phosphorylation
Journal of Neuroscience·2007·287 cit.·DOI
9
Expression of the Long Non-Coding RNA HOTAIR Correlates with Disease Progression in Bladder Cancer and Is Contained in Bladder Cancer Patient Urinary Exosomes
PLoS ONE·2016·274 cit.·DOI
10
Senescence-associated heterochromatin foci are dispensable for cellular senescence, occur in a cell type- and insult-dependent manner and follow expression of p16<sup>ink4a</sup>
Cell Cycle·2011·269 cit.·DOI

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