Cell Signaling Technology

Cyclin B1 (D5C10) Rabbit Monoclonal Antibody

#12231 · $161

100
Citing papers
5.7k
Combined citations
Supplier page Find this product

Research areas

Cancer-related Molecular Pathways12Ubiquitin and proteasome pathways9RNA Research and Splicing8RNA modifications and cancer8Cancer, Hypoxia, and Metabolism7PI3K/AKT/mTOR signaling in cancer7Metabolism, Diabetes, and Cancer7Autophagy in Disease and Therapy6

Papers citing this product

1
AMP-activated Protein Kinase and the Regulation of Autophagic Proteolysis
Journal of Biological Chemistry·2006·453 cit.·DOI
2
Extracellular Vesicles Derived from Bone Marrow Mesenchymal Stem Cells Protect against Experimental Colitis via Attenuating Colon Inflammation, Oxidative Stress and Apoptosis
PLoS ONE·2015·280 cit.·DOI
3
Inhibition of AMP-Activated Protein Kinase Signaling Alleviates Impairments in Hippocampal Synaptic Plasticity Induced by Amyloid β
Journal of Neuroscience·2014·195 cit.·DOI
4
The Role of AMPK and mTOR in Nutrient Sensing in Pancreatic β-Cells
Journal of Biological Chemistry·2007·178 cit.·DOI
5
Long non-coding RNA ZFAS1 interacts with CDK1 and is involved in p53-dependent cell cycle control and apoptosis in colorectal cancer
Oncotarget·2015·177 cit.·DOI
6
Synaptic Activity Controls Dendritic Spine Morphology by Modulating eEF2-Dependent BDNF Synthesis
Journal of Neuroscience·2010·158 cit.·DOI
7
Anthocyanin-rich fractions from red raspberries attenuate inflammation in both RAW264.7 macrophages and a mouse model of colitis
Scientific Reports·2014·152 cit.·DOI
8
Targeted Silencing of Elongation Factor 2 Kinase Suppresses Growth and Sensitizes Tumors to Doxorubicin in an Orthotopic Model of Breast Cancer
PLoS ONE·2012·118 cit.·DOI
9
1-Benzyl-3-cetyl-2-methylimidazolium iodide (NH125) Induces Phosphorylation of Eukaryotic Elongation Factor-2 (eEF2)
Journal of Biological Chemistry·2011·117 cit.·DOI
10
Anti-tumor efficacy of Selinexor (KPT-330) in gastric cancer is dependent on nuclear accumulation of p53 tumor suppressor
Scientific Reports·2018·105 cit.·DOI

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