Proteintech

MYC tag Polyclonal antibody

#16286-1-AP

71

Evidence Score · Well-Established

Used by 83 independent labs across 204 papers. How we score this →

200

Citing papers

2.9k

Combined citations

Supplier page Find this product

Research areas

Ubiquitin and proteasome pathways24 interferon and immune responses16 RNA modifications and cancer11 Autophagy in Disease and Therapy11 Ferroptosis and cancer prognosis9 Cancer-related molecular mechanisms research8 Cancer-related gene regulation8 Endoplasmic Reticulum Stress and Disease8

Papers citing this product

1

Circular RNA hsa_circ_0004872 inhibits gastric cancer progression via the miR-224/Smad4/ADAR1 successive regulatory circuit

Molecular Cancer·2020·239 cit.·DOI

2

CircNR3C2 promotes HRD1-mediated tumor-suppressive effect via sponging miR-513a-3p in triple-negative breast cancer

Molecular Cancer·2021·126 cit.·DOI

3

METTL3 acetylation impedes cancer metastasis via fine-tuning its nuclear and cytosolic functions

Nature Communications·2022·101 cit.·DOI

4

Deubiquitinase USP35 restrains STING-mediated interferon signaling in ovarian cancer

Cell Death and Differentiation·2020·89 cit.·DOI

5

USP25 regulates Wnt signaling by controlling the stability of tankyrases

Genes & Development·2017·76 cit.·DOI

6

Endonuclease G promotes autophagy by suppressing mTOR signaling and activating the DNA damage response

Nature Communications·2021·73 cit.·DOI

7

CHEK1 and circCHEK1_246aa evoke chromosomal instability and induce bone lesion formation in multiple myeloma

Molecular Cancer·2021·72 cit.·DOI

8

Deubiquitinase PSMD14 promotes ovarian cancer progression by decreasing enzymatic activity of PKM2

Molecular Oncology·2021·69 cit.·DOI

9

BUB1B and circBUB1B_544aa aggravate multiple myeloma malignancy through evoking chromosomal instability

Signal Transduction and Targeted Therapy·2021·68 cit.·DOI

10

The E3 ligase RNF5 restricts SARS-CoV-2 replication by targeting its envelope protein for degradation

Signal Transduction and Targeted Therapy·2023·61 cit.·DOI

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