Cell Signaling Technology

NLRP6 (E2S8A) Rabbit Monoclonal Antibody

#10650 · $175

74
Citing papers
1.8k
Combined citations
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Research areas

Galectins and Cancer Biology10Signaling Pathways in Disease7Peptidase Inhibition and Analysis6Cancer-related Molecular Pathways5Ginger and Zingiberaceae research4Adenosine and Purinergic Signaling4Autophagy in Disease and Therapy4Glycosylation and Glycoproteins Research4

Papers citing this product

1
Ca2+ Signaling but Not Store-Operated Ca2+ Entry Is Required for the Function of Macrophages and Dendritic Cells
The Journal of Immunology·2015·120 cit.·DOI
2
Macrophage migration inhibitory factor inhibition as a novel therapeutic approach against triple-negative breast cancer
Cell Death and Disease·2020·101 cit.·DOI
3
Galectin-3 binds to CD45 on diffuse large B-cell lymphoma cells to regulate susceptibility to cell death
Blood·2012·96 cit.·DOI
4
6-Shogaol Induces Apoptosis in Human Hepatocellular Carcinoma Cells and Exhibits Anti-Tumor Activity In Vivo through Endoplasmic Reticulum Stress
PLoS ONE·2012·92 cit.·DOI
5
A novel mechanism of autophagic cell death in dystrophic muscle regulated by P2RX7 receptor large-pore formation and HSP90
Autophagy·2015·82 cit.·DOI
6
A Disintegrin and Metalloprotease 17 Dynamic Interaction Sequence, the Sweet Tooth for the Human Interleukin 6 Receptor
Journal of Biological Chemistry·2014·73 cit.·DOI
7
Impact of Exogenous Galectin-9 on Human T Cells
Journal of Biological Chemistry·2015·70 cit.·DOI
8
Downregulating CD26/DPPIV by apigenin modulates the interplay between Akt and Snail/Slug signaling to restrain metastasis of lung cancer with multiple EGFR statuses
Journal of Experimental & Clinical Cancer Research·2018·68 cit.·DOI
9
Acetylsalicylic acid and salicylic acid present anticancer properties against melanoma by promoting nitric oxide-dependent endoplasmic reticulum stress and apoptosis
Scientific Reports·2020·60 cit.·DOI
10
Galectin‐9 mediates neutrophil capture and adhesion in a CD44 and β2 integrin‐dependent manner
The FASEB Journal·2021·55 cit.·DOI

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