Cell Signaling Technology

Ubc9 Antibody

#4918 · $317

100
Citing papers
8.7k
Combined citations
Supplier page Find this product

Research areas

Cancer, Hypoxia, and Metabolism20RNA modifications and cancer7Cancer-related Molecular Pathways6Ubiquitin and proteasome pathways6Metabolism, Diabetes, and Cancer6RNA Research and Splicing5Autophagy in Disease and Therapy5PI3K/AKT/mTOR signaling in cancer5

Papers citing this product

1
Activation of the hypoxia-inducible factor-1α pathway accelerates bone regeneration
Proceedings of the National Academy of Sciences·2008·516 cit.·DOI
2
LncRNA GLCC1 promotes colorectal carcinogenesis and glucose metabolism by stabilizing c-Myc
Nature Communications·2019·427 cit.·DOI
3
Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress
Science·2013·410 cit.·DOI
4
HIF-1α-induced expression of m6A reader YTHDF1 drives hypoxia-induced autophagy and malignancy of hepatocellular carcinoma by promoting ATG2A and ATG14 translation
Signal Transduction and Targeted Therapy·2021·378 cit.·DOI
5
Mitochondrial Phosphoenolpyruvate Carboxykinase Regulates Metabolic Adaptation and Enables Glucose-Independent Tumor Growth
Molecular Cell·2015·291 cit.·DOI
6
Glycolytic suppression dramatically changes the intracellular metabolic profile of multiple cancer cell lines in a mitochondrial metabolism-dependent manner
Scientific Reports·2019·283 cit.·DOI
7
BCR-signalling synergizes with TLR-signalling for induction of AID and immunoglobulin class-switching through the non-canonical NF-κB pathway
Nature Communications·2012·241 cit.·DOI
8
MicroRNA-141 enhances anoikis resistance in metastatic progression of ovarian cancer through targeting KLF12/Sp1/survivin axis
Molecular Cancer·2017·154 cit.·DOI
9
Tumour heterogeneity poses a significant challenge to cancer biomarker research
British Journal of Cancer·2017·147 cit.·DOI
10
Extracellular Signal-regulated Kinase (ERK)-dependent Gene Expression Contributes to L1 Cell Adhesion Molecule-dependent Motility and Invasion
Journal of Biological Chemistry·2004·142 cit.·DOI

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