Cell Signaling Technology

GFP Antibody

#2555 · $314

100
Citing papers
15.5k
Combined citations
Supplier page Find this product

Research areas

MicroRNA in disease regulation7RNA modifications and cancer7Cancer-related molecular mechanisms research6Extracellular vesicles in disease6RNA Research and Splicing6RNA and protein synthesis mechanisms5NF-κB Signaling Pathways5Genomics and Chromatin Dynamics5

Papers citing this product

1
The crucial role of protein phosphorylation in cell signaling and its use as targeted therapy (Review)
International Journal of Molecular Medicine·2017·1.3k cit.·DOI
2
Exosome engineering for efficient intracellular delivery of soluble proteins using optically reversible protein–protein interaction module
Nature Communications·2016·574 cit.·DOI
3
Nuclear condensates of the Polycomb protein chromobox 2 (CBX2) assemble through phase separation
Journal of Biological Chemistry·2018·359 cit.·DOI
4
Bone Morphogenetic Protein-2 Stimulates Runx2 Acetylation
Journal of Biological Chemistry·2006·343 cit.·DOI
5
Long Noncoding RNA H19 Promotes Osteoblast Differentiation Via TGF-β1/Smad3/HDAC Signaling Pathway by Deriving miR-675
Stem Cells·2015·311 cit.·DOI
6
Macrophage‐Disguised Manganese Dioxide Nanoparticles for Neuroprotection by Reducing Oxidative Stress and Modulating Inflammatory Microenvironment in Acute Ischemic Stroke
Advanced Science·2021·307 cit.·DOI
7
Neurodegenerative influence of oxidative stress in the retina of a murine model of diabetes
Diabetologia·2010·289 cit.·DOI
8
Neutralization of Transthyretin Reverses the Neuroprotective Effects of Secreted Amyloid Precursor Protein (APP) in APP<sub>Sw</sub>Mice Resulting in Tau Phosphorylation and Loss of Hippocampal Neurons: Support for the Amyloid Hypothesis
Journal of Neuroscience·2004·278 cit.·DOI
9
Hypoxia adipose stem cell-derived exosomes promote high-quality healing of diabetic wound involves activation of PI3K/Akt pathways
Journal of Nanobiotechnology·2021·270 cit.·DOI
10
Mitochondrial dysfunction promotes the transition of precursor to terminally exhausted T cells through HIF-1α-mediated glycolytic reprogramming
Nature Communications·2023·257 cit.·DOI

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