Cell Signaling Technology

CYP17A1 (E6Y3S) Rabbit Monoclonal Antibody

#10443 · $161,351

65
Evidence Score · Well-Established
Used by 270 independent labs across 332 papers. How we score this →
96
Citing papers
3.5k
Combined citations
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Research areas

Connective Tissue Growth Factor Research14interferon and immune responses9Cancer-related Molecular Pathways5melanin and skin pigmentation5Cytokine Signaling Pathways and Interactions5MicroRNA in disease regulation5Immune Cell Function and Interaction4Cholesterol and Lipid Metabolism4

Papers citing this product

1
EBV and human microRNAs co‐target oncogenic and apoptotic viral and human genes during latency
The EMBO Journal·2012·289 cit.·DOI
2
Connective Tissue Growth Factor Causes Glaucoma by Modifying the Actin Cytoskeleton of the Trabecular Meshwork
American Journal Of Pathology·2012·207 cit.·DOI
3
CCN2 Is Necessary for Adhesive Responses to Transforming Growth Factor-β1 in Embryonic Fibroblasts
Journal of Biological Chemistry·2006·149 cit.·DOI
4
Ectopic expression of microRNA-155 enhances innate antiviral immunity against HBV infection in human hepatoma cells
Virology Journal·2011·136 cit.·DOI
5
PINK1-Parkin Pathway Activity Is Regulated by Degradation of PINK1 in the Mitochondrial Matrix
PLoS Genetics·2014·131 cit.·DOI
6
The matrix protein CCN1 (CYR61) induces apoptosis in fibroblasts
The Journal of Cell Biology·2005·129 cit.·DOI
7
14-3-3ε and ζ Regulate Neurogenesis and Differentiation of Neuronal Progenitor Cells in the Developing Brain
Journal of Neuroscience·2014·128 cit.·DOI
8
Pancreatic cell plasticity and cancer initiation induced by oncogenic Kras is completely dependent on wild-type PI 3-kinase p110α
Genes & Development·2014·127 cit.·DOI
9
miR-155 induction in microglial cells suppresses Japanese encephalitis virus replication and negatively modulates innate immune responses
Journal of Neuroinflammation·2014·123 cit.·DOI
10
MicroRNA-15b Modulates Japanese Encephalitis Virus–Mediated Inflammation via Targeting RNF125
The Journal of Immunology·2015·107 cit.·DOI

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