Cell Signaling Technology

Total Ki-67 Matched Antibody Pair

#30420 · $1,726

34
Citing papers
2.1k
Combined citations
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Research areas

Immune Cell Function and Interaction8NF-κB Signaling Pathways6Occupational and environmental lung diseases4MicroRNA in disease regulation4Extracellular vesicles in disease4T-cell and B-cell Immunology4Immune cells in cancer3Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis3

Papers citing this product

1
A distinct and unique transcriptional program expressed by tumor-associated macrophages (defective NF- B and enhanced IRF-3/STAT1 activation)
Blood·2005·679 cit.·DOI
2
Notch1 augments NF‐κB activity by facilitating its nuclear retention
The EMBO Journal·2005·299 cit.·DOI
3
Potent Anti-Inflammatory Activity of Ursolic Acid, a Triterpenoid Antioxidant, Is Mediated through Suppression of NF-κB, AP-1 and NF-AT
PLoS ONE·2012·258 cit.·DOI
4
Exosomes with low miR-34c-3p expression promote invasion and migration of non-small cell lung cancer by upregulating integrin α2β1
Signal Transduction and Targeted Therapy·2020·128 cit.·DOI
5
MUC1 Protein Expression in Tumor Cells Regulates Transcription of Proinflammatory Cytokines by Forming a Complex with Nuclear Factor-κB p65 and Binding to Cytokine Promoters
Journal of Biological Chemistry·2011·95 cit.·DOI
6
NADH oxidase-dependent CD39 expression by CD8+ T cells modulates interferon gamma responses via generation of adenosine
Nature Communications·2015·78 cit.·DOI
7
Despite Inhibition of Nuclear Localization of NF-κB p65, c-Rel, and RelB, 17-β Estradiol Up-Regulates NF-κB Signaling in Mouse Splenocytes: The Potential Role of Bcl-3
The Journal of Immunology·2007·65 cit.·DOI
8
T Cell–Specific Notch Inhibition Blocks Graft-versus-Host Disease by Inducing a Hyporesponsive Program in Alloreactive CD4+ and CD8+ T Cells
The Journal of Immunology·2013·55 cit.·DOI
9
MicroRNAs of bone marrow mesenchymal stem cell-derived exosomes regulate acute myeloid leukemia cell proliferation and apoptosis
Chinese Medical Journal·2020·51 cit.·DOI
10
Nuclear factor-kappaB sensitizes to benzyl isothiocyanate-induced antiproliferation in p53-deficient colorectal cancer cells
Cell Death and Disease·2014·44 cit.·DOI

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