Cell Signaling Technology

LDHA/LDHC (C28H7) Rabbit Monoclonal Antibody

#3558 · $158

100
Citing papers
11.9k
Combined citations
Supplier page Find this product

Research areas

interferon and immune responses9Epigenetics and DNA Methylation7Cytokine Signaling Pathways and Interactions7PI3K/AKT/mTOR signaling in cancer6Cancer-related gene regulation6Viral Infections and Vectors5Immune Cell Function and Interaction5Cancer, Hypoxia, and Metabolism4

Papers citing this product

1
Active-Site Inhibitors of mTOR Target Rapamycin-Resistant Outputs of mTORC1 and mTORC2
PLoS Biology·2009·1.1k cit.·DOI
2
Orally bioavailable small-molecule inhibitor of transcription factor Stat3 regresses human breast and lung cancer xenografts
Proceedings of the National Academy of Sciences·2012·347 cit.·DOI
3
Curcumin Prevents High Fat Diet Induced Insulin Resistance and Obesity via Attenuating Lipogenesis in Liver and Inflammatory Pathway in Adipocytes
PLoS ONE·2012·298 cit.·DOI
4
S100B and S100A6 Differentially Modulate Cell Survival by Interacting with Distinct RAGE (Receptor for Advanced Glycation End Products) Immunoglobulin Domains
Journal of Biological Chemistry·2007·262 cit.·DOI
5
Stress Acts Cumulatively To Precipitate Alzheimer's Disease-Like Tau Pathology and Cognitive Deficits
Journal of Neuroscience·2011·248 cit.·DOI
6
TNF-α Downregulates Inhibitory Neurotransmission through Protein Phosphatase 1-Dependent Trafficking of GABA <sub>A</sub> Receptors
Journal of Neuroscience·2013·237 cit.·DOI
7
Inhibition of iNOS as a novel effective targeted therapy against triple-negative breast cancer
Breast Cancer Research·2015·221 cit.·DOI
8
The docking protein Gab1 is the primary mediator of EGF-stimulated activation of the PI-3K/Akt cell survival pathway
BMC Biology·2004·210 cit.·DOI
9
Activation of tyrosine kinase c-Abl contributes to α-synuclein–induced neurodegeneration
Journal of Clinical Investigation·2016·208 cit.·DOI
10
Transitional changes in the CRP structure lead to the exposure of proinflammatory binding sites
Nature Communications·2017·207 cit.·DOI

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