Cell Signaling Technology

S100A9 (D3U8M) Rabbit Monoclonal Antibody

#73425 · $169,369

66
Evidence Score · Well-Established
Used by 260 independent labs across 281 papers. How we score this →
22
Citing papers
570
Combined citations
Supplier page Find this product

Research areas

S100 Proteins and Annexins7Renal Diseases and Glomerulopathies4Immune Response and Inflammation3Sepsis Diagnosis and Treatment3Chronic Kidney Disease and Diabetes2Cancer Cells and Metastasis2Cell Adhesion Molecules Research2Immune cells in cancer2

Papers citing this product

1
Generative deep learning enables the discovery of a potent and selective RIPK1 inhibitor
Nature Communications·2022·98 cit.·DOI
2
Single-cell transcriptome profiling of sepsis identifies HLA-DRlowS100Ahigh monocytes with immunosuppressive function
Military Medical Research·2023·88 cit.·DOI
3
Stratification of radiosensitive brain metastases based on an actionable S100A9/RAGE resistance mechanism
Nature Medicine·2022·82 cit.·DOI
4
Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism
International Journal of Molecular Sciences·2023·68 cit.·DOI
5
Separation of breast cancer and organ microenvironment transcriptomes in metastases
Breast Cancer Research·2019·47 cit.·DOI
6
The TLR4-MyD88-NF-κB pathway is involved in sIgA-mediated IgA nephropathy
Journal of Nephrology·2020·31 cit.·DOI
7
Ze‐Qi‐Tang Formula Induces Granulocytic Myeloid‐Derived Suppressor Cell Apoptosis via STAT3/S100A9/Bcl‐2/Caspase‐3 Signaling to Prolong the Survival of Mice with Orthotopic Lung Cancer
Mediators of Inflammation·2021·18 cit.·DOI
8
Pentoxifylline Attenuates Proteinuria in Anti-Thy1 Glomerulonephritis via Downregulation of Nuclear Factor-κB and Smad2/3 Signaling
Molecular Medicine·2015·17 cit.·DOI
9
S100a8 silencing attenuates inflammation, oxidative stress and apoptosis in BV2 cells induced by oxygen‑glucose deprivation and reoxygenation by upregulating GAB1 expression
Molecular Medicine Reports·2020·16 cit.·DOI
10
The soluble VEGF receptor sFlt-1 contributes to endothelial dysfunction in IgA nephropathy
PLoS ONE·2020·16 cit.·DOI

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