Cell Signaling Technology

Mena Antibody

#2075 · $317

65
Evidence Score · Well-Established
Used by 191 independent labs across 202 papers. How we score this →
100
Citing papers
22.4k
Combined citations
Supplier page Find this product

Research areas

MicroRNA in disease regulation8Mesenchymal stem cell research6Telomeres, Telomerase, and Senescence6Nerve injury and regeneration5Immune Cell Function and Interaction5Epigenetics and DNA Methylation5Autophagy in Disease and Therapy5Cancer-related Molecular Pathways5

Papers citing this product

1
PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy
The Journal of Cell Biology·2010·1.9k cit.·DOI
2
The crucial role of protein phosphorylation in cell signaling and its use as targeted therapy (Review)
International Journal of Molecular Medicine·2017·1.3k cit.·DOI
3
Loss of epigenetic information as a cause of mammalian aging
Cell·2023·623 cit.·DOI
4
The Mammalian Target of Rapamycin Signaling Pathway Mediates Epileptogenesis in a Model of Temporal Lobe Epilepsy
Journal of Neuroscience·2009·517 cit.·DOI
5
Histamine Derived from Probiotic Lactobacillus reuteri Suppresses TNF via Modulation of PKA and ERK Signaling
PLoS ONE·2012·484 cit.·DOI
6
Proteome-wide identification of <i>in vivo</i> targets of DNA damage checkpoint kinases
Proceedings of the National Academy of Sciences·2007·417 cit.·DOI
7
A Mass Spectrometry-based Proteomic Approach for Identification of Serine/Threonine-phosphorylated Proteins by Enrichment with Phospho-specific Antibodies
Molecular & Cellular Proteomics·2002·385 cit.·DOI
8
Inhibitor-Sensitive FGFR1 Amplification in Human Non-Small Cell Lung Cancer
PLoS ONE·2011·369 cit.·DOI
9
Polyunsaturated fatty acids suppress glycolytic and lipogenic genes through the inhibition of ChREBP nuclear protein translocation
Journal of Clinical Investigation·2005·314 cit.·DOI
10
De novo lipogenesis in human fat and liver is linked to ChREBP-β and metabolic health
Nature Communications·2013·298 cit.·DOI

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