Decoding the SHOX2 methylation-expression paradox in lung adenocarcinoma: Dual-regulatory methylation patterns drive oncogenic activation and prognostic relevance
Yang Zhou(Chinese Academy of Medical Sciences & Peking Union Medical College), Jie He(Chinese Academy of Medical Sciences & Peking Union Medical College), Fucun Xie(Chinese Academy of Medical Sciences & Peking Union Medical College), Xiaoxiang Zhou(Chinese Academy of Medical Sciences & Peking Union Medical College), Guang Chen(HKU-Pasteur Research Pole), Zhao Li(Chinese Academy of Medical Sciences & Peking Union Medical College), Qi Xue(Chinese Academy of Medical Sciences & Peking Union Medical College), Ruochuan Zang(Chinese Academy of Medical Sciences & Peking Union Medical College), Zheng Zhang(Chinese Academy of Medical Sciences & Peking Union Medical College), Juhong Wang(Chinese Academy of Medical Sciences & Peking Union Medical College)
Cited by 0
Related Papers
METTL3 promotes tumour development by decreasing APC expression mediated by APC mRNA N6-methyladenosine-dependent YTHDF binding
|Nature Communications|2021|193
WNT/β-catenin-suppressed FTO expression increases m6A of c-Myc mRNA to promote tumor cell glycolysis and tumorigenesis
|Cell Death and Disease|2021|171
Acetate reprogrammes tumour metabolism and promotes PD-L1 expression and immune evasion by upregulating c-Myc
|Nature Metabolism|2024|100
A non‐metabolic function of hexokinase 2 in small cell lung cancer: promotes cancer cell stemness by increasing USP11‐mediated CD133 stability
|Cancer Communications|2022|84
Multimodal analysis of cell-free DNA whole-methylome sequencing for cancer detection and localization
|Nature Communications|2023|68