A mammalian, glutaminase-free asparaginase enhances venetoclax activity in preclinical AML models with chromosome 7 deletion
Dhabya Majid(The University of Texas MD Anderson Cancer Center), Hussein A. Abbas(The University of Texas MD Anderson Cancer Center)
Cited by 0
Related Papers
A cellular hierarchy framework for understanding heterogeneity and predicting drug response in acute myeloid leukemia
|Nature Medicine|2022|338
Cross-species identification of genomic drivers of squamous cell carcinoma development across preneoplastic intermediates
|Nature Communications|2016|159
Single cell T cell landscape and T cell receptor repertoire profiling of AML in context of PD-1 blockade therapy
|Nature Communications|2021|129