Inhibiting stearoyl‐ <scp>CoA</scp> desaturase suppresses bone metastatic prostate cancer by modulating cellular stress, <scp>mTOR</scp> signaling, and <scp>DNA</scp> damage response
Alexis Wilson(Wayne State University), Izabela Podgorski(The Barbara Ann Karmanos Cancer Institute)
Cited by 1
Related Papers
Bone marrow adipocytes promote tumor growth in bone via FABP4-dependent mechanisms
|Oncotarget|2013|210
Bone marrow adipocytes promote the Warburg phenotype in metastatic prostate tumors<i>via</i>HIF-1α activation
|Oncotarget|2016|123
Adipocyte-activated oxidative and ER stress pathways promote tumor survival in bone via upregulation of Heme Oxygenase 1 and Survivin
|Scientific Reports|2018|42
New 3D-Culture Approaches to Study Interactions of Bone Marrow Adipocytes with Metastatic Prostate Cancer Cells
|Frontiers in Endocrinology|2016|38
Targeting SCD in SCD-amplified prostate cancer inhibits growth in bone by modulating cellular stress, mTOR, and DNA damage pathways
|bioRxiv (Cold Spring Harbor Laboratory)|2025|0