Dual NLRC4 and non-canonical inflammasome signaling drives human GSDMD-mediated killing of <i>Shigella flexneri</i> independently of bacterial cardiolipin
Mateusz Szczerba(Harvard University), Marcia B. Goldberg(Broad Institute)
Cited by 0
Related Papers
Longitudinal proteomic analysis of severe COVID-19 reveals survival-associated signatures, tissue-specific cell death, and cell-cell interactions
|Cell Reports Medicine|2021|309
Disruption of IcsP, the major <i>Shigella</i> protease that cleaves IcsA, accelerates actin‐based motility
|Molecular Microbiology|1997|132
The unipolar <i>Shigella</i> surface protein IcsA is targeted directly to the bacterial old pole: IcsP cleavage of IcsA occurs over the entire bacterial surface
|Molecular Microbiology|1999|121
Plasma proteomics reveals tissue-specific cell death and mediators of cell-cell interactions in severe COVID-19 patients
|bioRxiv (Cold Spring Harbor Laboratory)|2020|49