Replication fork stalling at DNA lesions is driven by competition between damage bypass pathways
Amir Aharoni(Ben-Gurion University of the Negev), Helle D. Ulrich(Institute of Molecular Biology)
Cited by 0
Related Papers
Spatial separation between replisome‐ and template‐induced replication stress signaling
|The EMBO Journal|2018|55
Avidity-based biosensors for ubiquitylated PCNA reveal choreography of DNA damage bypass
|Science Advances|2023|6
The ubiquitin-conjugating enzyme Ubc13-Mms2 cooperates with a family of FYVE-type-RING ubiquitin protein ligases in K63-polyubiquitylation at internal membranes
|bioRxiv (Cold Spring Harbor Laboratory)|2019|0