Replication fork stalling at DNA lesions is driven by competition between damage bypass pathways
Amir Aharoni(Ben-Gurion University of the Negev), Helle D. Ulrich(Institute of Molecular Biology)
Cited by 0
Related Papers
Spatial separation between replisome‐ and template‐induced replication stress signaling
|The EMBO Journal|2018|55
Point Mutation of a Non-Elastase-Binding Site in Human α1-Antitrypsin Alters Its Anti-Inflammatory Properties
|Frontiers in Immunology|2018|9
Avidity-based biosensors for ubiquitylated PCNA reveal choreography of DNA damage bypass
|Science Advances|2023|6
Application of directed evolution and back-to-consensus algorithms to human alpha1-antitrypsin leads to diminished anti-protease activity and augmented anti-inflammatory activities
|Cellular Immunology|2020|4
The ubiquitin-conjugating enzyme Ubc13-Mms2 cooperates with a family of FYVE-type-RING ubiquitin protein ligases in K63-polyubiquitylation at internal membranes
|bioRxiv (Cold Spring Harbor Laboratory)|2019|0