Nicorandil attenuates thioacetamide induced liver fibrosis via AMPK, SIRT1 and HIF1α mediated cellular energy homeostasis
Magy R. Kozman(Misr University for Science and Technology), Ahmed M. Atwa(Thi Qar University), Yasmine H. Ahmed(Cairo University), Fatma El-Zahraa S. Abdel Rahman(Nahda University), Mohamed G. Ewees(The Ohio State University), Saad Misfer Alqahtani(Najran University), Yasmin M. Ahmed(Cairo University), Ehab A. M. El‐Shoura(Al-Azhar University), Lamiaa Khalaf Ahmed(Al-Azhar University), Omar A. Farghaly(Al-Azhar University), Mostafa Sabry(Al-Azhar University)
Cited by 3
Related Papers
Long-term electronic cigarette exposure induces cardiovascular dysfunction similar to tobacco cigarettes: role of nicotine and exposure duration
|American Journal of Physiology-Heart and Circulatory Physiology|2021|74
Chronic cigarette smoke exposure triggers a vicious cycle of leukocyte and endothelial-mediated oxidant stress that results in vascular dysfunction
|American Journal of Physiology-Heart and Circulatory Physiology|2020|63
Electronic cigarette exposure causes vascular endothelial dysfunction due to NADPH oxidase activation and eNOS uncoupling
|American Journal of Physiology-Heart and Circulatory Physiology|2022|63
Effect of scopoletin on phagocytic activity of U937-derived human macrophages: Insights from transcriptomic analysis
|Genomics|2020|55
Cytoglobin has potent superoxide dismutase function
|Proceedings of the National Academy of Sciences|2021|48