The Proinflammatory Secretome of Senescent Cells Can Be Controlled by a <scp>HIF2A</scp> ‐Dependent Upregulation and a <scp>FURIN</scp> ‐Dependent Cleavage of the <scp>ANGPTL4</scp> Secreted Factor
Gabriela Makulyte(Centre National de la Recherche Scientifique), David Bernard(University College London)
Cited by 0
Related Papers
Myc confers androgen-independent prostate cancer cell growth
|Journal of Clinical Investigation|2003|195
PLA2R1 Mediates Tumor Suppression by Activating JAK2
|Cancer Research|2013|75
Lysyl oxidase family activity promotes resistance of pancreatic ductal adenocarcinoma to chemotherapy by limiting the intratumoral anticancer drug distribution
|Oncotarget|2016|74
Transcriptional repression of DNA repair genes is a hallmark and a cause of cellular senescence
|Cell Death and Disease|2018|66
Targeting chemoresistant senescent pancreatic cancer cells improves conventional treatment efficacy
|Molecular Biomedicine|2023|27