CHK1 Alleviates Doxorubicin-Induced Cardiotoxicity via Activating AKT/mTOR Pathway in Mice
Haiyang Ni(Wuhan University), See PDF, Yulan Ma(Wuhan University), Qizhu Tang(Wuhan University), Teng Teng(Wuhan University)
Cited by 0
Related Papers
FNDC5 alleviates oxidative stress and cardiomyocyte apoptosis in doxorubicin-induced cardiotoxicity via activating AKT
|Cell Death and Differentiation|2019|452
Metabolic landscape in cardiac aging: insights into molecular biology and therapeutic implications
|Signal Transduction and Targeted Therapy|2023|169
CTRP3 attenuates cardiac dysfunction, inflammation, oxidative stress and cell death in diabetic cardiomyopathy in rats
|Diabetologia|2017|157
CTRP3 protected against doxorubicin-induced cardiac dysfunction, inflammation and cell death via activation of Sirt1
|Journal of Molecular and Cellular Cardiology|2017|146
Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways
|EBioMedicine|2017|139