DNMT3A-R882 mutations intrinsically drive dysfunctional neutropoiesis from human haematopoietic stem cells
Giovanna Mantica(University of Cambridge), Elisa Laurenti(Wellcome/MRC Cambridge Stem Cell Institute), Noa Chapal(Weizmann Institute of Science), Aleksandra Krzywon(University of Cambridge), George S. Vassiliou(Wellcome/MRC Cambridge Stem Cell Institute), Matthew Collin(Centre for Human Genetics), Mark D. Minden(University Health Network), Eoin McKinney(University of Cambridge), Peter J. Campbell(Wellcome Sanger Institute), Liran I. Shlush(Maccabi Healthcare Services), Hector Huerga Encabo(Universitat Pompeu Fabra), Amos Tuval(Weizmann Institute of Science), Michele Marongiu(Institute of Genetic and Biomedical Research), Francesco Cucca(University of Sassari), William G. Dunn(Wellcome/MRC Cambridge Stem Cell Institute), Dominique Bonnet(The Francis Crick Institute), Margarete A. Fabre(University of Cambridge), Daniel Hayler(Wellcome/MRC Cambridge Stem Cell Institute), Antonella Santoro(University of Cambridge), Valeria Orrù(Institute of Genetic and Biomedical Research), Kendig Sham(University of Cambridge), Hugo Bastos(University of Cambridge), Tamir Biezuner(Weizmann Institute of Science), Andrea Arruda(University Health Network), Adi Danin(Weizmann Institute of Science), Jyoti Nangalia(Wellcome Sanger Institute), Joe Lee(Wellcome Sanger Institute), Edoardo Fiorillo(Institute of Genetic and Biomedical Research), Nick Williams(Wellcome Sanger Institute), Yoni Moskovitz(Weizmann Institute of Science), Emily Mitchell(Wellcome Sanger Institute), Aditi Vedi(Wellcome/MRC Cambridge Stem Cell Institute)
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