KRAS-induced STN1 (OBFC1) promotes proper CTC1–STN1–TEN1 complex-independent DNA double-strand break repair and cell cycle checkpoint maintenance in pancreatic cancer
Changxian Shen(The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research Institute), Terence M. Williams(City Of Hope National Medical Center)
Cited by 2
Related Papers
Oncogenic KRAS drives radioresistance through upregulation of NRF2-53BP1-mediated non-homologous end-joining repair
|Nucleic Acids Research|2021|68
Regulation of CHK1 by mTOR contributes to the evasion of DNA damage barrier of cancer cells
|Scientific Reports|2017|47
P53 suppresses ribonucleotide reductase via inhibiting mTORC1
|Oncotarget|2017|38
Targeting oncogenic KRAS in non-small cell lung cancer with EGFR aptamer-conjugated multifunctional RNA nanoparticles
|Molecular Therapy — Nucleic Acids|2023|24