Metabolic modulation of pyruvate dehydrogenase and glucose oxidation in diabetic cardiomyopathy: pathways, perspectives, and potentials
Jordan S. F. Chan(University of Alberta), John R. Ussher(University of Alberta), Linyue Dong(University of Alberta)
American Journal of Physiology-Heart and Circulatory Physiology
September 15, 2025
Cited by 0
Related Papers
Increased ketone body oxidation provides additional energy for the failing heart without improving cardiac efficiency
|Cardiovascular Research|2019|195
GIPR Is Predominantly Localized to Nonadipocyte Cell Types Within White Adipose Tissue
|Diabetes|2022|62
Cardiac-Specific Deletion of Pyruvate Dehydrogenase Impairs Glucose Oxidation Rates and Induces Diastolic Dysfunction
|Frontiers in Cardiovascular Medicine|2018|42
The antianginal ranolazine mitigates obesity-induced nonalcoholic fatty liver disease and increases hepatic pyruvate dehydrogenase activity
|JCI Insight|2019|18
Ketone ester administration improves glycemia in obese mice
|American Journal of Physiology-Cell Physiology|2023|18