The E3 ligase HECTD4 regulates COX-2-dependent tumor progression and metastasis
Joanna Vuille(Harvard University), Daniel A. Haber(Howard Hughes Medical Institute), J. Keith Joung(Massachusetts General Hospital), Wilhelm Haas(Harvard University), Douglas B. Fox(Harvard University), Sambhavi Animesh(Center for Cancer Research), Ji Eun Kwak(Harvard University), Julian Grünewald(Massachusetts General Hospital), Andrew E. H. Elia(Massachusetts General Hospital), Ben K. Wesley(Center for Cancer Research), Eugene Oh(Center for Cancer Research), Regan N. Szalay(Harvard University), Ezgi Antmen(Harvard University), Hunter C. Russell(Harvard University), D. Gulhan(Harvard University), Ben S. Wittner(Broad Institute), Cem Tanrıöver(Center for Cancer Research), Shyamala Maheswaran(Massachusetts General Hospital), Eric Zaniewski(Center for Cancer Research), Soroush Hajizadeh(Massachusetts General Hospital), Min Yang(Shandong University), Richard Y. Ebright(Harvard University), Douglas S. Micalizzi(Massachusetts General Hospital), Zachary J. Nicholson(Center for Cancer Research), Robert Morris(Massachusetts General Hospital)
Cited by 1
Related Papers
Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1
|Nature|2009|4.2k
Isolation of rare circulating tumour cells in cancer patients by microchip technology
|Nature|2007|3.5k
High-frequency off-target mutagenesis induced by CRISPR-Cas nucleases in human cells
|Nature Biotechnology|2013|3.3k
CRISPR-Cas systems for editing, regulating and targeting genomes
|Nature Biotechnology|2014|3.2k
Efficient genome editing in zebrafish using a CRISPR-Cas system
|Nature Biotechnology|2013|3k