Mitochondrial dysfunction mediated by ER-calcium dysregulation in neurons derived from Alzheimer’s disease patients
Sarah Mustaly-Kalimi(Rosalind Franklin University of Medicine and Science), Grace E. Stutzmann(Rosalind Franklin University of Medicine and Science), Wacey Gallegos(Rosalind Franklin University of Medicine and Science), Daniel Steinbrenner(Rosalind Franklin University of Medicine and Science), Robert A. Marr(Rosalind Franklin University of Medicine and Science), Smriti Gupta(University of Virginia), Aiden Houcek(Rosalind Franklin University of Medicine and Science), Israel Sekler(Ben-Gurion University of the Negev), Daniel A. Peterson(Rosalind Franklin University of Medicine and Science), David A. Bennett(Rush University Medical Center)
Cited by 16
Related Papers
NIA‐AA Research Framework: Toward a biological definition of Alzheimer's disease
|Alzheimer s & Dementia|2018|10.4k
Toward defining the preclinical stages of Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
|Alzheimer s & Dementia|2011|7.1k
A/T/N: An unbiased descriptive classification scheme for Alzheimer disease biomarkers
|Neurology|2016|1.9k
REST and stress resistance in ageing and Alzheimer’s disease
|Nature|2014|784
NCLX is an essential component of mitochondrial Na <sup>+</sup> /Ca <sup>2+</sup> exchange
|Proceedings of the National Academy of Sciences|2009|776