GRK-biased adrenergic agonists for the treatment of type 2 diabetes and obesity
Aikaterini Motso(Stockholm University), Tore Bengtsson(Stockholm University)
Cited by 19
Related Papers
β-Adrenoceptors, but not α-adrenoceptors, stimulate AMP-activated protein kinase in brown adipocytes independently of uncoupling protein-1
|Diabetologia|2005|98
α1A-Adrenoceptors Activate Glucose Uptake in L6 Muscle Cells through a Phospholipase C-, Phosphatidylinositol-3 Kinase-, and Atypical Protein Kinase C-Dependent Pathway
|Endocrinology|2004|53
β2-Adrenergic activation increases glycogen synthesis in L6 skeletal muscle cells through a signalling pathway independent of cyclic AMP
|Diabetologia|2006|36
BRL37344 stimulates GLUT4 translocation and glucose uptake in skeletal muscle via β<sub>2</sub>-adrenoceptors without causing classical receptor desensitization
|American Journal of Physiology-Regulatory, Integrative and Comparative Physiology|2019|20