PROS1‐MERTK Axis Drives Tumor Microenvironment Crosstalk and Progression in Papillary Thyroid MicrocarcinomaWenqian Zhang(Tangshan Gongren Hospital), Wei Sun(First Hospital of China Medical University)Advanced ScienceMay 28, 202510.1002/advs.202413474Cited by 4SaveCiteExport RISWatch citationsRelated PapersNEAT1_2 functions as a competing endogenous RNA to regulate ATAD2 expression by sponging microRNA-106b-5p in papillary thyroid cancer|Cell Death and Disease|2018|85Astrocyte-derived exosomes protect hippocampal neurons after traumatic brain injury by suppressing mitochondrial oxidative stress and apoptosis|Aging|2021|83NEAT1_2/RRAD/EHF Positive Feedback Loop Facilitates Aerobic Glycolysis in Papillary Thyroid Cancer Cells|Endocrinology|2023|12