Clinically translatable mitochondrial gene therapy in muscle using tandem mtZFN architecture
P. Nash(University of Cambridge), Michal Minczuk(MRC Mitochondrial Biology Unit)
Cited by 7
Related Papers
Mutations in GTPBP3 Cause a Mitochondrial Translation Defect Associated with Hypertrophic Cardiomyopathy, Lactic Acidosis, and Encephalopathy
|The American Journal of Human Genetics|2014|157
TEFM variants impair mitochondrial transcription causing childhood-onset neurological disease
|Nature Communications|2023|30
mtFociCounter for automated single-cell mitochondrial nucleoid quantification and reproducible foci analysis
|Nucleic Acids Research|2023|3
mtFociCounter for automated single-cell mitochondrial nucleoid quantification and reproducible foci analysis
|bioRxiv (Cold Spring Harbor Laboratory)|2022|0
mtFociCounter for automated single-cell mitochondrial nucleoid quantification and reproducible foci analysis
|Zenodo (CERN European Organization for Nuclear Research)|2023|0