AMPK-dependent Parkin activation suppresses macrophage antigen presentation to promote tumor progression
Xinyu Wang(Sun Yat-sen University), Jun Chen(Harbin Institute of Technology), Juanjuan Ou(Army Medical University), Jing Liao(Guangzhou Medical University), Jean‐François Trempe(McGill University Health Centre), Edward A. Fon(Montreal Neurological Institute and Hospital), Di Yu(East China Jiaotong University), Yingzhao Wang(Sun Yat-sen University), Zijin Xia(Sun Yat-sen University), Lixiang Wang(Sun Yat-sen University), Congzhou Luo(Sun Yat-sen University), Yi Wei(Regeneron (United States)), Hongrui Song(Sun Yat-sen University), Chengzhou Mao(Shenzhen University), Yongyi Feng(Sun Yat-sen University), Yujia Liu(Sun Yat-sen University), Jingjing Duan(Sun Yat-sen University), Limin Zheng(Weifang University of Science and Technology), W.F. Mai(Sun Yat-sen University), Chunliu Huang(Sun Yat-sen University), Hai Hong(Sun Yat-sen University), Cheng Zhong(Second Affiliated Hospital of Zhejiang University)
Cited by 12
Related Papers
Ubiquitin is phosphorylated by PINK1 to activate parkin
|Nature|2014|1.5k
Low-dose interleukin-2 treatment selectively modulates CD4+ T cell subsets in patients with systemic lupus erythematosus
|Nature Medicine|2016|570
Preparation of Hierarchical Hollow CaCO<sub>3</sub> Particles and the Application as Anticancer Drug Carrier
|Journal of the American Chemical Society|2008|453
Intratumoral dendritic cell–CD4+ T helper cell niches enable CD8+ T cell differentiation following PD-1 blockade in hepatocellular carcinoma
|Nature Medicine|2023|389
<scp>USP</scp> 8 regulates mitophagy by removing <scp>K</scp> 6‐linked ubiquitin conjugates from parkin
|The EMBO Journal|2014|367