Targeting aldolase A in hepatocellular carcinoma leads to imbalanced glycolysis and energy stress due to uncontrolled FBP accumulation
Marteinn T. Snaebjornsson(German Cancer Research Center), Almut Schulze(German Cancer Research Center), Ramona Rudalska(University of Tübingen), Felix C. E. Vogel(German Cancer Research Center), Alina M. Winkelkotte(German Cancer Research Center), Roberto Carlos Frias-Soler(German Cancer Research Center), Ralf Steuer(Humboldt-Universität zu Berlin), Florian Röhrig(University of Würzburg), Lisa Schlicker(German Cancer Research Center), Philipp Biechl(German Cancer Research Center), Jessica Denise Schwarz(University of Würzburg), Daniel Dauch(Bernstein Center for Computational Neuroscience Tübingen), Elmar Wolf(Hochschule für Angewandte Wissenschaften Kiel), Daria Komkova(Humboldt-Universität zu Berlin), Kamal M. Al-Shami(German Cancer Research Center), Ioanna Koltsaki(German Cancer Research Center), Adriano B. Chaves‐Filho(German Cancer Research Center)
Cited by 30
Related Papers
Senescence surveillance of pre-malignant hepatocytes limits liver cancer development
|Nature|2011|1.6k
Lipid metabolism in cancer
|FEBS Journal|2012|1.3k
FOXO3a regulates reactive oxygen metabolism by inhibiting mitochondrial gene expression
|Cell Death and Differentiation|2011|312